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Orbital

Thyroid eye disease, eyelid retraction & orbital decompression

A complete pathway for thyroid eye disease, from medical management of active inflammation to orbital decompression, squint surgery and eyelid surgery in the rehabilitation phase.

Summary

Thyroid eye disease (TED) is an autoimmune inflammation of the eye-socket tissues, most commonly seen in patients with Graves’ disease. It causes the eye to bulge forward, the eyelids to retract, the eye to be red and swollen, and (in severe cases) double vision and loss of vision from optic-nerve compression.

I lead the regional service for orbital decompression at Portsmouth Hospitals University NHS Trust, and manage TED across all stages, in close collaboration with endocrinology and orthoptic colleagues.

Who is this for?

I see patients across the full TED pathway:

  • newly diagnosed patients with mild–moderate inflammation,
  • those with active disease being considered for steroids or immunomodulation,
  • patients with sight-threatening optic-nerve compression needing urgent decompression,
  • patients in the rehabilitation phase requiring decompression, squint or eyelid surgery,
  • patients with stable disease and persistent eyelid retraction (the ‘staring’ appearance).

What the procedure involves

Treatment is staged across the disease cycle:

  1. Active phase: smoking cessation, selenium, lubrication, and (where indicated) intravenous steroids or immunomodulation, in close partnership with endocrinology.
  2. Orbital decompression: surgical removal of bone (medial wall, floor, and/or lateral wall) and fat to give the eye room to recede. Performed via a hidden incision and/or transconjunctival approach.
  3. Squint (strabismus) surgery: for stable double vision, in collaboration with strabismus colleagues.
  4. Eyelid surgery: including upper- and lower-lid recession to address eyelid retraction, and blepharoplasty for residual fullness.

The order is important. Decompression is performed first, then squint surgery, then eyelid surgery, because each can affect the next.

Recovery and what to expect

  • Decompression: overnight stay, marked bruising and swelling for 1–2 weeks, full recovery over 6–8 weeks.
  • Eyelid recession: day case, 1–2 weeks of swelling.
  • Most patients describe a transformative improvement in both comfort and appearance.

Risks and alternatives

Decompression is a major operation. Risks include bleeding, infection, sinus problems, double vision (around 20–30% of patients require subsequent squint surgery), numbness of the cheek, and very rarely loss of vision.

Non-surgical alternatives (lubrication, prisms, botulinum toxin to the upper-lid retractor) have a role in mild cases and as bridging measures.

Frequently asked

Common questions.

I have an overactive thyroid. Does that mean I will get eye disease?

Most patients with thyroid disease never develop significant eye involvement. Smoking is the largest modifiable risk factor; stopping smoking is the single most important step you can take.

When is decompression done?

Most decompressions are performed once the active inflammatory phase has settled (around 6–18 months). Urgent decompression is sometimes needed for sight-threatening compression of the optic nerve.

Is teprotumumab available in the UK?

Teprotumumab is approved in the US and is becoming available in the UK on a case-by-case basis. NHS access is currently limited; the rehabilitative surgical pathway remains the established treatment.

Book a consultation

Make an enquiry.

Private consultations are arranged through Gina Stacey, my secretary. NHS appointments are by GP referral via Portsmouth Hospitals University NHS Trust.

Private secretary
Gina Stacey

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NHS

NHS appointments at Queen Alexandra Hospital are arranged via your GP or optometrist through Portsmouth Hospitals University NHS Trust.

In an emergency

For urgent eye problems please call NHS 111, attend the on-call eye casualty service, or call 999 if it is life-threatening.

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